Are you interested in using your lived experience and voice to help us shape the future pulmonary fibrosis research?
This online webinar has now taken place.
A recording of the event is available on the MTWC Seminar Series webpage.
The MTWC lecture series is primarily aimed at researchers and healthcare professionals, which means that elements of each lecture are likely to be very technical. However, interested members of the public are welcome to attend. Read on to find out more about what Dr Adam Byrne will talk about during his MTWC lecture.
The role of macrophage metabolism in regulating the airways
Cellular metabolism is the process by which the cells of the body burn nutrients in order to carry out their basic functions. Immuno-metabolism is an emerging field that investigates the interaction between the immune system and cellular metabolism. Interest in this field is gaining momentum due to the realisation that changes in how cells utilise energy underlies many immune responses, and that manipulating cellular metabolism can increase or reduce inflammation.
Airway macrophages are key immune cells which regulate the lung environment.
In this talk I will discuss my groups work which explores how cellular metabolism is linked to airway macrophage function and how these processes are linked to chronic lung diseases such as asthma and idiopathic pulmonary fibrosis.
Dr Adam Byrne Biography
"I completed my undergraduate studies in Chemistry at University College Dublin and subsequently undertook a PhD in medicinal chemistry at the School of Pharmacy and Pharmaceutical Sciences, Trinity College Dublin. After a period in industry, I carried out postdoctoral work at Northwestern University, Chicago; this work focused on immune pathways involved in asthma, allergy and anaphylaxis. In 2011, I joined Imperial College London to carry further postdoctoral work, focusing on immune responses during lung fibrosis. I was appointed as a Lecturer in Chronic Lung Disease in 2016. My work focuses on the role on airway macrophage ontogeny and metabolism during chronic lung diseases such as asthma and idiopathic pulmonary fibrosis."
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